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By Jeffrey C. Hall

The sphere of genetics is quickly evolving and new scientific breakthroughs are occuring because of advances in wisdom won from genetics learn. This sequence constantly publishes vital experiences of the broadest curiosity to geneticists and their colleagues in affiliated disciplines.

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Extra resources for Advances in Genetics, Vol. 62

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M. (2008). Letting the genome out of the bottle—will we get our wish? N. Engl. J. Med. 358, 105–107. Hunter, D. , Jacobs, K. , Cox, D. , Hankinson, S. , et al. (2007a). A genome-wide association study identifies alleles in FGFR2 associated with risk of sporadic postmenopausal breast cancer. Nat. Genet. 39, 870–874. Hunter, D. , Hoover, R. , and Chanock, S. J. (2007b). Scanning the horizon: What is the future of genome-wide association studies in accelerating discoveries in cancer etiology and prevention?

Lander, E. , and Altshuler, D. (2002). Human genome sequence variation and the influence of gene history, mutation and recombination. Nat. Genet. 32, 135–142. , Milne, R. , Fernandez, L. , et al. (2006). Evaluating HapMap SNP data transferability in a large-scale genotyping project involving 175 cancerassociated genes. Hum. Genet. 118, 669–679. Richards, R. , and Sutherland, G. R. (1993). Fragile X syndrome unstable element, p(CCG)n, and other simple tandem repeat sequences are binding sites for specific nuclear proteins.

1993) have been added over time. 3; Wilson, 1994, 1998). The Framingham investigators have assigned “weights” to these risk factors and have developed “prediction scores” to predict the development of CHD (Truett et al. 1967). , 2001). 4. III. PHENOTYPIC AND GENETIC ARCHITECTURE OF COMPLEX TRAITS Biological variation may be understood at two levels: phenotypic and genetic. Many of the CVD risk factors such as HDL-cholesterol (HDL-C), total cholesterol, and blood pressure are quantitative traits.

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